Aberration Dmel\Df(1)r-D1
| General Information | |||
|---|---|---|---|
| Symbol | Dmel\Df(1)r-D1 | Species | D. melanogaster |
| Name | Deficiency (1) rudimentary | FlyBase ID | FBab0000884 |
| Feature type | chromosomal_deletion | Created / Updated | 2006-08-22/2006-08-22 |
| Formalized genetic data | mei-41 << bk1 << l(1)15DFb << l(1)15DFa << bk2 << l(1)ESHS51 | ||
| Sequence coordinates | |||
| Deleted segment | 14C5--15B1 | ||
| Duplicated segment | |||
| Computed Breakpoints include | 14C5-14C6;15B1 | ||
| Breakpoints Inherited | |||
Nature of the Aberration
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| Cytological Order | |||
| Progenitor | |||
| Mutagen | |||
| Class of aberration (relative to progenitor) | |||
| Breakpoints | 14C2-14C4;15B2-15C1 14B;15B 14C5-14C6;15A6-15B1 14B6;15A2 14D1-14D2;15D1-15D2 | ||
| Causes alleles | |||
| Carries alleles | |||
| Transposon Insertions | |||
| Genetic mapping information | |||
| Comments | |||
Comments on Cytology
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Limits of break 1 from polytene analysis (FBrf0079877) Left limit of break 2 from polytene analysis (FBrf0076870) Right limit of break 2 from polytene analysis (FBrf0079877) Simple deficiency. Breakpoints are 14B6;15A2 or 14C2-14C4;15B2 Ref: FBrf0040979. | |||
Molecularly Mapped Breakpoints
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Sequence Crossreferences
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| DDBJ
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EMBL / GenBank | DNA sequence Protein sequence Name | ||
Gene Deletion & Duplication Data
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Genes Deleted / Disrupted
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| Complementation Data | |||
| Completely deleted / disrupted | |||
| Molecular Data | |||
Genes NOT Deleted / Disrupted
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| Complementation Data | |||
| Molecular Data | |||
Genes Duplicated
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| Complementation Data | |||
| Molecular Data | |||
Genes NOT Duplicated
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| Complementation Data | |||
| Molecular Data | |||
Related Comments
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Phenotypic Data
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| In combination with other aberrations | |||
| NOT in combination with other aberrations | Hemizygous embryos were examined with polarised light microscopy and antibody staining: no syncytial muscles develop, embryos are immobile, and visceral mesoderm development is also impaired. Heterozygosity for this deletion has no effect on the mutant ovarian phenotype of ovoD2. Homozygous embryos are very abnormal compared to wild-type. Intermediate second site non-complementing phenotype with zipEbr and zipmhc-c6.1: malformed phenotype penetrance 25-75%. Midgut development of mutant embryos is wild type. Semilethal opposite r, r29, or r38 para- r- Two Minutes surround r ; the one on the left, M(1)14C, is new, the one on the right is RpS5 (Lefevre) | ||
Position Effect Variegation Data
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Stocks
( 3 ) | |||
| Kyoto | |||
| Bloomington | |||
Notes on Origin
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| Discoverer | |||
Balancer / Genotype Variants of the Aberration
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Separable Components
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Other Comments
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"14C5--6;15A6--B1" was stated as revision. | |||
Synonyms & Secondary IDs
( 8 ) | |||
| Reported As | |||
| Symbol Synonym | Df(1)(D) Df(1)r1D Df(1)rD1 Df(1)rD1 Df(1)r-D1 Df(1)r-Dl | ||
| Name Synonym | Deficiency (1) rudimentary | ||
| Secondary FlyBase IDs | |||
References
( 27 ) | |||
| Generate a list of | |||
| List References by type |
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Recent research papers (0)
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| All research papers listed in FlyBase were published before 2006 | |||
Nature of the Aberration